Acta Neurochirurgica Supplements by Andrea Lavinio, Bogdan Ene-Iordache, Ilaria Nodari, Alan

By Andrea Lavinio, Bogdan Ene-Iordache, Ilaria Nodari, Alan Girardini, Elena Cagnazzi (auth.), H. -J. Steiger (eds.)

86 brief papers originating from the thirteenth overseas Symposium on Intracranial strain and mind tracking held in July 2007 in San Francisco current experimental in addition to scientific learn information on invasive and non-invasive intracranial strain and mind biochemistry tracking. The papers have passed through a peer-reviewing and are prepared in 8 sections: mind damage: ICP administration and cerebral body structure; hydrocephalus and cerebrospinal fluid dynamics; complex neuromonitoring; biomedical informatics; imaging; ICP: mind compliance, biophysics, and biomechanics; stroke, subarachnoid hemorrhage, and intracerebral hematoma; and experimental experiences and types. The papers handle the expanding use of decompressive craniectomy for the therapy of mind edema in addition after mind damage and the swiftly increasing box of complex neuromonitoring and neuroimaging.

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This is also supported by the correlation between PI and lac-JV in the present study. Most patients with severe head injury have increased lactate production, as measured by microdialysis [3]. In our study, high levels of lac-JV were observed before HBO treatment. Increased lactate production indicates an anaerobic metabolic status caused by a lack of oxygen and mitochondria damage [1]. Cerebrospinal fluid lactate levels were consistently decreased by HBO treatment [10]. Mean brain tissue lactate levels decreased by 40% in oxygen-treated patients who suffered from head injury [5].

For the ICP recordings of a plateau wave and intractable intracranial hypertension, changes in the values of HMF of both models and the corresponding changes in simulated arterial–arteriolar bed resistance are shown in Fig. 2. The derived HMF values of the numerical system identification model and the corresponding HMF values of the biomechanical model increased at the onset of the plateau wave during vasodilation and decreased at termination during the constrictive phase of the wave (see Fig. 2a).

Stroke 29:159–165 2. Balestreri M, Czosnyka M, Steiner LA, Schmidt E, Smielewski P, Matta B, Pickard JD (2004) Intracranial hypertension: what additional information can be derived from ICP waveform after head injury? Acta Neurochir (Wien) 146:131–141 3. Balestreri M, Steiner LA, Czosnyka M (2003) Sex-related differences and traumatic brain injury. J Neurosurg 99(3):616 4. Bramlett HM, Dietrich WD (2001) Neuropathological protection after traumatic brain injury in intact female rats versus males or ovariectomized females.

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